The Seventh Generation Guide to a Toxin-Free Home Part 1: Understanding what’s toxic Introduction Few products typify American consumerism as well as household cleaners. Capitalizing onour insecurities, manufacturers and marketers have transformed a mundane collection ofproducts into over an $18 billion market of household helpers. We’re constantly told we’llhumiliate ourselves if our toilet bowls and counter tops don’t sparkle as well as ourneighbors’ do.Marketing hyperbole aside, modern cleaners are significantly more effective than theirpredecessors. Synthetic cleaning agents, anti-redeposition agents, bleaches, builders, enzymes and optical brighteners have produced a generation of products that work under more varied conditions, against more forms of dirt, in colder water, and with less time and effort than ever before. But in our attempts to get our clothes whiter than white and homes cleaner than clean, we’ve accepted a plethora of chemicals whose presence in our homes raises very serious health and environmental concerns.
What happens when I use traditional cleaning products? More than you might realize! Today’s cleaning products are made from an eye-opening number of surprisingly toxic chemicals. When we use these products in our homes, the chemicals they contain can stay suspended in the air for hours or even days after the product has been used and can easily be inhaled. These chemicals also remain behind as residues on surfaces to which the cleaners have been applied. In this way, they can be easily absorbed through any skin that comes into contact with those surfaces. In addition, when chemicals from different cleaners accidentally come into contact with each other, they sometimes react to form new toxic substances. Or this mixing can magnify the potential health effects that are caused by either or both of the chemicals alone. The results of all this chemical chaos can be deadly. A 15 year study in Oregon, comparing women who didn’t work outside the home with women who did, found a 54% higher death rate from cancer in the women who stayed at home. The study suggested that chronic exposure to cleaning products played a role.Each year there are 5 to 10 million household product poisonings reported—mostly of children. With all these chemicals in our homes, it’s no wonder that the EPA found the air quality in our homes to be 5 to 10 times more toxic than the air outside and typically contaminated by anywhere between 20 to 150 different pollutants in concentrations 10 to 40 times those outdoors. Much of this pollution comes from petrochemical cleaners. Don’t product labels warn meabout hazardous ingredients? Unfortunately, the answer is no. Though cleaners are the only household products regulated by the Consumer Product Safety Commission under the Federal Hazardous Substances Labeling Act, they’re not required to reveal their ingredients. These ingredients are considered “trade secrets” and government regulations are designed to protect this proprietary information rather than human health or the environment. In short, no one but cleaner manufacturers really know exactly what is in these products. The consumer has little to go on beyond the warning labels manufacturers are required to put on their products. Though mandated signal words like DANGER, WARNING and CAUTION give us a very general idea about the overall seriousness of the unknown substances the products contain, they do little more than that. In fact, a New York Poison Control Center study found 85% of product warning labels to be inadequate. Furthermore, these warnings only apply to the immediate health effects a product causes and don’t address what really happens when we use these cleaners regularly in our homes. When is something toxicand when is it not? An examination of the issue of hazardous chemicals hiding in common household products starts with this simple question. And the answer may surprise you because the toxic potential of any given material is not so much a matter of what it’s made from but rather how much of it to which you areexposed. For example, during the 18th century, a pale complexion was considered attractive and a sign of good breeding. Tanning salons were definitely “out.” To achieve their pallor, the members of King Louis XVI’s court took arsenic, perhaps weekly. Although we consider arsenic to be highly toxic, neither King Louis nor his wife, Marie Antoinette, died of arsenic poisoning. In fact, some level of arsenic in the diet is still considered necessary for good health! In contrast, many beneficial chemicals have caused death. Aspirin, one of the safest and most versatile medicines known, poisoned countless children before packaging laws were enacted. Table salt is a common part of our daily diet, and an adult would have to ingest close to a half cup (400 grams) to receive a fatal dose. Yet, an accidental substitution of salt for lactose in baby formulas has caused fatal poisoning.What, then, makes a chemical a poison? One answer is quantity (acute toxicity). Another is time (chronic toxicity). When it comes to acute toxicity (or sudden death from exposure to a chemical), it is the amount needed to induce sudden death that determines whether a chemical is considered poisonous or not.Safe doses are measured by a statistical standard known as Lethal Dose (LD). The LD standard is a useful tool in determining the toxicity of a particular chemical, but isunfortunately largely derived from tests conducted on animals.(Because this issue is important to us, we’d like to pause here to note that Seventh Generation neither conducts nor approves of animal testing under any circumstances. We believe there are better and far more humane ways to measure toxicity, and we employ these alternative methods when testing our own products. However, both the scientific community and the cleaning products industry as a whole rely on the LD standard almost exclusively, a fact which means that no one has ever created an alternative set of similarly comprehensive, animal testing-free data. Because the LD standard is the only way to illustrate several crucial points, we’re forced to use it here in spite of our reservations. The good news is that this will only take a moment or two.) The LD standard is based on a benchmark called the LD50. The LD50 is the quantity of a chemical needed to kill 50% of the animals in a test group (usually mice or rats). Because larger animals require larger doses of a chemical to exhibit toxic effects (i.e., it takes more arsenic to kill an elephant than a mouse), the LD50 is measured as the weight of chemical in milligrams (or mg) per kilogram (or kg) of animalweight needed to cause death. For example, the LD50 of arsenic trioxide (a common form of arsenic), when measured in rats, is 15 mg/kg. This means about 15 mg (approximately one-half of one-thousandth of an ounce, or 0.0005 ounces) would be needed to kill a 1 kilogram (2.2 pound) rat. By comparison, 3,000 mg (approximately a tenth of an ounce, or 0.1 ounce) wouldbe needed to kill a 200 kg (440 pound) gorilla. The LD50 of aspirin, measured in rats, is 1,500 mg/kg. This means 1,500 mg (0.05 ounce) would be needed to kill a 1 kg rat, and 300,000 mg (10 ounces, over half a pound) would be needed to kill the 200 kg gorilla. The LD50 of table salt (also measured in rats) is 3,750 mg/kg. At this rate, it would take 750,000 mg (nearly a pound and a half!) of salt to kill the same gorilla.What’s important to note is that it takes 100 times more aspirin to show acutely toxic effects in a given animal than arsenic trioxide. In other words, arsenic trioxide is 100 times more toxic than aspirin. It takes more than twice as much salt to kill an animal as aspirin. Thus, salt is less than half as toxic as aspirin. Confused? Don’t be. Just remember that almost everything is poisonous in some amount. The less of a chemical that’s needed to show acutely toxic effects, the more poisonous it is.Aside from ingestion, other forms of acute toxicity that must be considered for consumer products include inhalation toxicity (especially for volatile, gaseous, and “dusty” substances) and dermal toxicity (for substances that contact our skin). The cancer/chemical connection: How little is little enough? Fortunately, we are seldom exposed to sufficiently large doses of chemicals to suffer acutely toxic effects. In most circumstances, a person is regularly exposed to a substance at levels significantly below the acutely toxic level. This is called chronic exposure. Tobacco smoke, present in many homes, contains many toxic chemicals. Most exposure to tobacco smoke does not result in instant mortality because the levels of exposure are below the acutely toxic level. Over time, though, toxic effects are experienced from tobacco smoke. The effects are most visible in smokers suffering emphysema; lung, nose and throat cancer; and other chronic ailments. Nonsmokers who live or work in smoke-filled environmentsalso suffer chronic effects. Most people who come into contact with the chemicals in our homes and environment do not experience acutely toxic exposure leading to sudden death. They are more likely to experience an array of far subtler symptoms, including headaches, rashes, nausea, and others, which, while less dramatic, can still be debilitating. Compounding this problem is the difficulty of isolating which chemical present in your home, office, or even car is causing the problem. Measuring cancer risk from chronic exposure to chemicals is no less difficult. The best data comes from occupational If the LD50 is: The CPSC Defines the Hazard as(product would also carry the notice): 5,000 mg/kg or higher UndefinedBetween 50 and 5,000 mg/kg Toxic (“Warning, Keep out of Reach of Children”) Less than 50 mg/kg Highly toxic (“Danger” “Poison”) (Note that by this definition both table salt and aspirin are considered toxic materials. Arsenic trioxide is highly toxic.)The Consumer Product Safety Commission (CPSC) defines acute oral toxicity as follows: chemical exposures that result in unique malignancies. For example, chimney sweeps in 19th Century England developed cancer of the scrotum much more frequently than the general population. We now know this was due to exposure topolynuclear aromatic hydrocarbons in the soot with which they had daily contact. Similarly, lung cancers in shipyardworkers implicated asbestos as a carcinogen, as did livercancers in workers manufacturing polyvinyl chloride (PVC). Incidence among polyvinyl chloride workers of this form of cancer is 3,000 times higher than among the generalpopulation. There are strong links between increased cancer rates and life in the industrialized world, where we are exposed to high levels of suspected cancer-causing chemicals. In Sandra Steingraber’s outstanding book Living Downstream (see Further Suggested Reading), she documents somepowerful information: • One-half of the world’s cancers occur among people in industrialized countries, even though we are only one-fifth of the population. • Breast cancer rates are 30 times higher in the United States than in parts of Africa. • The International Agency for Research on Cancer has concluded that 80% of all cancer is attributable to environmental influences (these include lifestyle influences such as smoking, as well as exposure to carcinogenic chemicals). • During our lifetime, 40% of all Americans will get some form of cancer—50% of men and 30% of women. Amazingly, only a dozen or so chemicals have been directly implicated in human cancers (for more information on why this is so, read Toxic Deception, listed in Further Suggested Reading). Most of the other “suspected” carcinogens have been identified by feeding large doses of these chemicals to specially bred mice and rats. If a chemical produces tumors in one or more feeding studies, it is only considered a suspectedcarcinogen. While many, many household chemicals fall into the category of “suspected carcinogen,” regulations that might protect us from them remain relatively few and far between. This is so for two reasons:First, it is difficult to apply the results of animal studies (which measure high levels of exposure for short periods of time) to real-world human exposures (which typically involve low levels of exposure for long periods of time). Because chemicals can cause different effects in the body depending on the dose and length of exposure, using short term animal studies to predict long term human outcomes is often an exercise in futility. Such studies simply don’t accurately reflect the way ordinary people actually use and are exposed to most chemicals. They do a good job of telling us what will happen when we experience a lot of exposure over a little time but not a little exposure over a lot of time. We may, for example, know if you ingest a pound of chemical X in a single sitting, you will sicken and die. But what happens when you’re exposed to just a few thousandths of a gram of chemical X every day for many, many years? The study that told us what will happen in the first case simply cannot predict what will happen in the second.Developing research methods that can accurately predict real-world human consequences of long term, low-level exposures to particular chemicals is an inherently daunting task for a simple reason: the longer the study period, the more potential risk factors are introduced. As time passes, it becomes harder and harder to say with certainty that chemical X is responsible for condition Y because so many other variables, identified and unidentified, have likely entered the picture and created health effects of their own that interfere with the study’s results. At a certain point, separating these unwanted factors and their effects from effects of the chemical one actually wanted to study in the first place becomes virtually impossible.There is also the very serious issue of research ethics. Irrefutable evidence of human health effects from exposure to specific chemicals can only truly come from one source: tests on human beings over long periods of time, and clearly such tests are out of the question.Because they cannot be conducted on humans and because they suffer from built-in imperfections, those studies that do attempt to gauge long term, real-world health effects are often easy to dispute, and this brings us to the second reason for the relative absence of strong consumer protections and other chemical regulations: the power of the chemical industry itself. Whenever the test results do manage to come close to suggesting a certain chemical is dangerous enough to be removed from the market, the chemical’s manufacturer is likely to spend millions of dollars challenging the research and any potential regulations based upon it. Take, for example, the case of dioxin. Industry lawyers and lobbyists have claimed that even though hundreds of tests and studies indicate that dioxin is a very probable cause of cancer, we still don’t know for sure because no actual tests were done on humans! The result is that while most of Europe is satisfied with this 99% level of certainty and has stopped bleaching paper with chlorine because of the dioxin the process creates, we continue to use chlorine here in the U.S. The 1% of uncertainty that remains has been enough to quell regulations here. In fact, the Chlorine Institute, an industry lobbying group, admits that it spends approximately $150 million a year fighting anyone and everyone who challenges the safety of this chemical!
Natural, organic and synthetic: What’s the difference? When it comes to understanding household chemicals, this is a crucial question, and a point about which people are often understandably confused. All matter in our universe is composed of atoms. There are approximately 110 types of atoms, or elements. Ninety-two elements occur naturally, and just 10 elements account for over 99% of the things we enjoy on Earth. One of those elements, carbon, is uniquely associated with life. Hence, chemical compounds containing carbon are called organic chemicals.In the 19th century, humankind began to make its own chemicals using carbon. Although they did not occur in nature, these human-made compounds of carbon were still called organic chemicals. They are “synthetic organic chemicals,” rather than “natural organic chemicals,” which is an important distinction.Synthetic organic chemicals: A short history, Part 1 Hundreds of millions of years ago, Earth was covered by oceans filled with millions of tons of tiny plants and animals. As these plants and animals died, they settled to the bottom of the oceans and were covered by thousands of feet of sediment and rock. Over millions of years, heat and pressure turned the layers of dead plants and animals into a viscous, black material we call petroleum or crude oil. Petroleum consists of many long chains of carbon atoms with hydrogen atoms attached. These long chains, called hydrocarbons, do not have much use. But when they are broken into shorter chains, we get materials like ethylene (a building block for synthetic detergents and plastics), propane and butane (petroleum gases used as fuel), gasoline, diesel fuel, heating oil, and lubricants. This process of breaking the long chains of petroleum into shorter chains is called cracking. Once petroleum has been cracked, all the products are jumbled together. They have to be separated, and this is done by boiling the mixture of chains. Because each product boils at a different temperature, it separates from the mixture at different times as the temperature of the boil gradually increases. Once released, the product, whether gasoline or ethylene, is captured and condensed back to a liquid state. This process is called distillation, and it produces surprisingly pure products called, cleverly, “petroleum distillates.” Petroleum distillates can be used without further processing. Liquid petroleum gas (LPG), gasoline, diesel fuel, and heating oil are petroleum distillates used to produce energy. Similar products, called naphthas, Stoddard solvents, or just plain old petroleum distillates, are used as solvents on greases and tars that will not dissolve in water.In addition to the toxic nature of the products petroleum produces, our reliance on this material causes a host of environmental problems in and of itself. Petroleum pollutes the environment when we drill for it, when we transport it (oil spills average 2.6 million gallons a month), and when we refine it (refineries release 492 million pounds of hazardous volatile organic compounds and over 71 million pounds of toxic air pollutants into our air and water each year). Every time we use a petrochemical cleaning product, we contribute to this pollution. And, we further deplete an important global resource whose supplies are expected to become scarce around the year 2050.
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